肺炎衣原體間接免疫熒光檢測(cè)試劑盒

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廣州健侖長(zhǎng)期供應(yīng)各種生物原料，主要代理品牌：美國(guó)Seracare、西班牙Certest、美國(guó)Fuller、美國(guó)NOVABIOS、 Cellabs等等。

Cellabs公司是一個(gè)的生物技術(shù)公司，總部位于澳大利亞悉尼。專門研發(fā)與生產(chǎn)針對(duì)熱帶傳染性疾病的免疫診斷試劑盒。其產(chǎn)品40多個(gè)國(guó)家和地區(qū)。1998年，Cellabs收購(gòu)TropBio公司，進(jìn)一步鞏固其在研制熱帶傳染病、寄生蟲診斷試劑方面的位置。

肺炎衣原體間接免疫熒光檢測(cè)試劑盒
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主要產(chǎn)品包括：隱孢子蟲診斷試劑，賈第蟲診斷試劑，瘧疾診斷試劑，衣原體檢測(cè)試劑，絲蟲診斷試劑，錐蟲診斷試劑等。

廣州健侖生物科技有限公司與cellabs達(dá)成代理協(xié)議，歡迎廣大用戶咨詢訂購(gòu)。

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【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
【騰訊 】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-3室
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由此說(shuō)明，紋狀體內(nèi)存在乙酰膽堿遞質(zhì)系統(tǒng)，其 功能的加強(qiáng)將導(dǎo)致震顫麻痹癥狀的出現(xiàn)?？偨Y(jié)以上的研究結(jié)果，目前 認(rèn)為黑質(zhì)上行抵達(dá)紋狀體的多巴胺遞質(zhì)系統(tǒng)的功能，在于抑制紋狀體 內(nèi)乙酰膽堿遞質(zhì)系統(tǒng)的功能；震顫麻痹患者由于多巴胺遞質(zhì)系統(tǒng)功能 受損，導(dǎo)致乙酰膽堿遞質(zhì)系統(tǒng)功能的亢進(jìn)，才出現(xiàn)一系列癥狀。如果 應(yīng)用左旋多巴以增強(qiáng)多巴胺的合成，或應(yīng)用M型受體阻斷劑以阻斷乙酰 膽堿的作用，均能治療震顫麻痹。在正常情況下，黑質(zhì)上行抵達(dá)紋狀 體的多巴胺遞質(zhì)系統(tǒng)還與行為覺(jué)醒有關(guān)。動(dòng)物實(shí)驗(yàn)觀察到，單純破壞 中腦黑質(zhì)多巴胺遞質(zhì)系統(tǒng)后，則動(dòng)物在行為上不能表現(xiàn)覺(jué)醒，對(duì)新異 的刺激不能表現(xiàn)探究行為。震顫麻痹患者面部表情呆板，可能就是行 為覺(jué)醒發(fā)生障礙的表現(xiàn)。舞蹈病患者的主要臨床表現(xiàn)為不自主的上肢 和頭部的舞蹈樣動(dòng)作，并伴有肌張力降低等。病理研究證明，患者紋 狀體嚴(yán)重萎縮，而黑質(zhì)-紋狀體通路是完好的。在這類患者，若采用左 旋多巴進(jìn)行治療反而使癥狀加劇，而用利血平耗竭多巴胺遞質(zhì)卻可使 癥狀緩解。神經(jīng)生化的研究發(fā)現(xiàn)，患者紋狀體中膽堿能神經(jīng)元和γ-氨 基丁酸能神經(jīng)元的功能明顯減退。因此認(rèn)為，舞蹈病病變主要是紋狀 體內(nèi)的膽堿能和γ-氨基丁酸能神經(jīng)元功能減退，而黑質(zhì)多巴胺能神經(jīng) 元功能相對(duì)亢進(jìn)，這和震顫麻痹的病變機(jī)制正好相反。目前知道，黑 質(zhì)和紋狀體之間是有環(huán)路的：黑質(zhì)的多巴胺能上行系統(tǒng)能抑制紋 狀體內(nèi)膽堿能和γ-氨基丁酸能系統(tǒng)的活動(dòng)；而紋狀體的γ-氨基丁酸 下行系統(tǒng)能反饋抑制黑質(zhì)的多巴胺能系統(tǒng)的活動(dòng)（圖11-16）。臨床治 療震顫麻痹時(shí)，如左旋多巴用得過(guò)量則可引起類似舞蹈病的癥狀，也 說(shuō)明上述的觀點(diǎn)是有道理的。殼核和尾狀核通過(guò)大量條紋狀細(xì)胞橋互 相連接，所以得名紋狀體。根據(jù)發(fā)生的早晚可分為新、舊紋狀體，新 紋狀體指豆?fàn)詈说臍ず臀矤詈?，舊紋狀體指蒼白球，紋狀體屬錐體外 系的結(jié)構(gòu)，與骨骼肌的活動(dòng)有關(guān)。 在發(fā)生學(xué)上比較年輕，包括尾狀核 及殼核，它們起源于端腦。在這兩個(gè)神經(jīng)細(xì)胞團(tuán)中，含有大量的小細(xì) 胞和較少的大細(xì)胞。
This shows that there is an acetylcholine transmitter system in the striatum, and its enhanced function will lead to the appearance of paralysis symptoms. Summing up the above research results, it is currently believed that the function of the dopamine transmitter system of the substantia nigra arriving at the striatum is to inhibit the function of the acetylcholine transmitter system in the striatum; in patients with tremor paralysis, the function of the dopamine transmitter system is impaired, resulting in the transmission of acetylcholine. Only when the quality of the system functions, can a series of symptoms appear. If levodopa is used to enhance dopamine synthesis, or if an M-type receptor blocker is used to block the effects of acetylcholine, tremor paralysis can be treated. Under normal conditions, the dopamine transmitter system that the substantia nigra arrives at the striatum is also associated with behavioral arousal. Animal experiments have observed that after simply destroying the dopamine neurotransmitter system of the substantia nigra, the animals cannot behave awake in their behavior, and they can't express inquiry behavior for novel stimuli. The facial expression of a paralyzed paralyzed patient may be a manifestation of an arousal disorder. The main clinical manifestations of patients suffering from chorea are involuntary dance-like movements of the upper limbs and head, accompanied by decreased muscle tone. Pathological studies have demonstrated that the patient's striatum is severely atrophied and the nigro-striatal pathway is intact. In these patients, treatment with levodopa may worsen the symptoms, while depletion of dopamine transmitters with reserpine can relieve symptoms. Neurochemical studies have shown that the function of cholinergic and γ-aminobutyric acid neurons in the striatum of patients is significantly reduced. Therefore, it is believed that choriopathic lesions are mainly cholinergic and gamma-aminobutyric acid neuronal dysfunction in the striatum, and the substantia nigra dopaminergic neuronal function is relatively hyperactive, which is contrary to the pathogenesis of tremor paralysis. It is currently known that there is a looping link between the substantia nigra and the striatum: the dopaminergic ascending system of the substantia nigra inhibits the activity of cholinergic and gamma-aminobutyric acid energy systems in the striatum; whereas striatal The down-stream system of gamma-aminobutyric acid can feedback inhibit the activity of the dopaminergic system of the substantia nigra (Figure 11-16). When clinical therapy is used to paralyze paralysis, the use of excessive amounts of levodopa may cause symptoms similar to those of chorea. This also makes sense. The putamen and caudate nucleus are connected to each other by a large number of striped cell bridges, so the striatum is named. According to the occurrence of early and late can be divided into new and old striatum, the new striatum refers to the lenticular nucleus of the shell and caudate nucleus, the old striatum refers to the globus pallidus, striatum is the extrapyramidal structure, and skeletal muscle Related activities. It is relatively young in herogenesis, including caudate nucleus and putamen, which originated in the encephalon. In these two nerve cell clusters, there are a large number of small cells and fewer large cells.